1. The molecular basis of chemoresistance is poorly understood, precluding advances in glioma treatment and leaving gliomas among the most lethal tumors.

2. A Brainstem glioma is a cancerous glioma tumor in the Brainstem

3. Malignant glioma is currently a fatal disease.

4. Brainstem gliomas start in the brain or spinal cord tissue and …

5. Objective To explore the effect of microsurgery on glioma.

6. Establish human glioma cell strains and culture normal astrocytes.

7. Objective : To improve the effect of diagnosis and therapy for insular gliomas.

8. Conclusion The intrinsic radiosensitivity varies in different human glioma cells lines.

9. P gp and LRP sometimes had co overexpression in glioma tissues.

10. Some Brainstem gliomas can be classified based on certain growth characteristics: Focal Brainstem gliomas: These tumors grow more slowly, and are restricted to one area of the Brainstem (usually the midbrain and medulla)

11. A low grade glioma was found, and he was treated with radiation.

12. 27 Purpose To observe the anti - glioma effects of lymphocyte activated by SEC.

13. Purpose To observe the anti - glioma effects of lymphocyte activated by SEC.

14. Brainstem glioma refer to all subtypes of astrocytomas that occur in the Brainstem

15. I have a 68-year-old female with an advanced brain stem glioma.

16. However, diagnostic accuracy is still insufficient in some cases, for instance in patients who have malignant gliomas.

17. Objective: To obtain GFP-RECK expression vector and to express it in glioma cell line SHG

18. Glioma, post-infection or other diagnosis was impressed. Suboccipital craniectomy with total tumor excision was performed.

19. What is glioBlastoma multiforme? GBM is a grade 4 glioma brain tumor arising from brain cells called glial cells

20. Altered expression of CCN3 has been observed in a variety of tumors, including hepatocellular carcinomas, Wilm's tumors, Ewing's sarcomas, gliomas, rhabdomyosarcomas, and adrenocortical carcinomas.

21. Objective:To explore the role and mechanism of suppression of ribonuclease RNase inhibitor in the growth of glioma cells.

22. Objective To investigate the cytotoxic effect of fraction C from Naja Naja atra venom (FC) on human glioma cell line (U2.

23. Our research group developed an oncolytic adenovirus that will soon enter a clinical phase I trial in patients diagnosed with glioma.

24. Brainstem gliomas can arise in any area of the Brainstem, but the pons is the most common location making up 75-85% of Brainstem tumor

25. The histologic determination of the degree of tissue anaplasia and grade of malignancy of gliomas is based upon qualitative histological features (nuclear pleomorphism, mitoses, endothelial proliferation, tumor necrosis).

26. Brainstem gliomas typically grow in a diffuse fashion among the normal cells within the Brainstem, making it impossible to remove the tumor without injuring the normal Brainstem.

27. CONCLUSION: The vasogenic brain edema induced by glioma cells may not be the result of hyperpermeability of blood-brain barrier to macromolecules in plasma.

28. Objectives: To study whether the C6 rat glioma cell line contains brain tumor stem-like cells and its growing pattern in serum-free medium supplemented with mitogens.

29. PTT efficacy was evaluated for both empty (Ma) and Auns-loaded Ma (Ma(NS)) in both monolayers and spheroids consisting of C6 rat glioma cells and Ma.

30. Actinium-225 and treatments derived from it have also been used in early trials for leukemia, melanoma, and glioma. But something stood in the way of expanding this treatment

31. Objective: The study is focus on the inhibition effect of natural plant Ficua carica L. (fig) on glioma, and explores the mechanisms of the effect in an experimental research.

32. This is particularly true for patients with newly diagnosed and relapsing glioblastoma and anaplastic glioma as well as the treatment of medulloblastoma and primary lymphoma of the central nervous system (CNS).

33. Gliolan should only be used by experienced brain surgeons who are familiar with surgery of malignant glioma, who have an in-depth knowledge of the anatomy of the brain, and who have completed a training course in surgery guided by fluorescence

34. Agog: Art Gallery of Golden (Golden, British Columbia, Canada) Agog: Australian Genomics and Clinical Outcomes of High Grade Glioma (Australia) Agog: Advisory Group on Governance (UK) Agog: Asian Gynecologic Oncology Group (Taiwan) Agog: Arizona Gun Owners Guide (Alan Korwin book) Agog: Alumni Geographers of Geneseo (est

35. Based on the profiles of aldolase isozyme, the tumours tested seemed to be classified into two groups: those with a higher activity of aldolase C in A-C set (astrocytomas such as mixed type with oligodendroglioma, plump cell type and somewhat anaplastic type) and those with a rather low to no detectable activity of aldolase C in A-C set (malignant glioma, neuroblastoma, neurinoma, meningioma, pituitary adenoma, pinealoma, craniopharyngioma, secondary carcinoma).

36. This review summarizes findings, discusses problems and defines new questions from the phase III trials on anaplastic gliomas. The molecular subgroup analyses of the NOA-04 trial identified three molecular parameters, which predict longer progression-free and overall survival independent from the mode of therapy, radiotherapy or alkylating chemotherapy-. These are 1p/19q codeletion, methylation of the promoter of the O6-methylguanyl methyltransferase (MGMT) gene and hot-spot mutations in the isocitrate dehydrogenase 1 (IDH1) gene.